ADC-1001 is a FIND® optimized interleukin-1 receptor antagonist (IL-1Ra)

with enhanced receptor affinity compared to the wild type IL-1Ra protein. It is suitable for the treatment of any IL-1-driven condition.

Alligator Bioscience is seeking partnership with pharmaceutical and biotech companies for the development and commercialization of the program. 


Interleukin-1 and interleukin-1 receptor antagonist

IL-1 is a pro-inflammatory cytokine and has been shown to play an important role in the pathogenesis in several inflammatory conditions. IL-1 exerts its inflammatory effect by interacting with its receptor, IL-1 receptor type I (IL-1RI). IL-1Ra is a natural antagonist of the IL-1RI capable of binding to IL-1RI without inducing cellular activation. Once bound to IL-1RI, IL-1Ra prevents binding of IL-1 to the receptor and thus works as a competitive antagonist.



Medical Need & Market

There is a great unmet medical need for several inflammatory conditions in which IL-1 has been shown to cause or contribute to disease pathogenesis. Some of these indications are rather common with prevalence of 0.5-1% (e.g. RA and gout) whereas others are orphan indications (e.g. juvenile arthritis and cryopyrin-associated periodic syndromes).


The ADC competitive advantage
 

ADC-1001 is expected to have increased potency compared to wild type IL-1Ra (Anakinra). In addition, the therapeutic efficacy of IL-1Ra is expected to be enhanced by an increased circulation half-life. Protein modifications increasing the half-life often result in loss of function. Therefore, ADC-1001 would be an optimal protein for half-life extension, due to its enhanced receptor affinity and inhibitory activity.


Scientific References

E. Dahlen, et al., "Development of interleukin-1 receptor antagonist mutants with enhanced antagonistic activity in vitro and improved therapeutic efficacy in collagen-induced arthritis," J Immunotoxicol. 5(2), 189 (2008).

ADC-1001: Interleukin-1 (IL-1) receptor antagonist

Mechanism:
Interleukin-1 (IL-1) receptor antagonist

Effects:
• Inhibition of IL-1 interaction with the
  IL-1 receptor 1
• Inhibition of pro-inflammatory cytokine
  production and immune activation
• Inhibition of cartilage and bone
  degradation

Profile:
•  2nd generation of Anakinra (Kineret®)
•  Increased receptor affinity compared
   to Anakinra
•  17.3 kD
•  Short circulation half-life
•  Subcutaneous injection

Potential Indications:
Autoimmune and inflammatory diseases
• Rheumatoid arthritis
• Juvenile arthritis
• Cryopyrin-associated periodic 
  syndromes
• Gout

Production:
Expressed in E.coli

IP:
Filed patent application: WO2008132485

Project status:
Pre-clinical

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